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MARKET RESEARCH INSIGHTS amp BUSINESS INTELLIGENCE 5 Most Effective Ways to Use Twitter for Market Research Jul 9, 2013 2:44:00 PM Social media is creating better opportunities to engage and share expertise, family photos, and self-aggrandizing quotes (ok, maybe Im just talking about myself, but you get the point.) Specifically, Twitter has empowered us to drop pint size knowledge in under 140 characters. Its popularity is increasing with each tweet. According to eBiz, Twitter is the 9th most visited website on the Inte. From teenagers to corporations, Twitters pration makes it an attractive option to conduct market research. Beyond the live micro-blogging capabilities, there are golden opportunities to obtain business intelligence from the Twittermunities. Here are the 5 most effective ways to use Twitter for market research: Person-to-personmunication: A market researcher can jump on their mobile app of choice and ask a question to their followers.

Within seconds, you can expect to receive feedback from people. Twitter removes the barriers that inhibited you from engaging with your market on a personal and low-energy basis. Stream of conscious monitoring: The beauty of Twitter lies in its simplicity and dynamism. This social natural observation allows you to deepen your understanding of consumer values and beliefs in a purely unfiltered way. According to Daze Info.

175 million tweets are sent daily. The data warehouse facilitated by Twitter is an analysts Shangri La. You can monitor the pulse of your customers and inform your qualititative analysis towards brand equity. Broadening your market research scope: The world is using social media at a rapidly increasing rate and on the go. According to Socialbankers, 66 of user-generated tweets mentioning brands are done via mobile devices. Your customers are discussing you all the time. Its your job to put yourself in the position to listen. Discover latent consumer insights: Its about what you dont know.

Ananizations downfall could invariably stem from failing to account for factors that they never knew. The rawmunication provided by Twitter gives exclusive access to how a customer really feels about you and even improves your responsiveness to customer issues. Learn your customers social media language: A major mistake that ananization can make is to expect the customer to learn how to talk to them. Social media is an arena where eachwork has its own tone and language. It behooves the brand to learn how to relate to the customer in their language to facilitate a positive relationship.

For more information on how social media can supplement your market reseach, follow us on Twitter. Thanks for reading Richard Washington Marketing Manager MarketResearch 3A2F2Fmarketresearch2Fimages2F1x1000000.gif /

SEE DOCUMENTARY The th About gs is a series of thirteen illustrated g information booklets containing facts about the mostmonly abused gs. This website contains the full text of these booklets, which were specifically written for young people, but contain facts adults need to know as well. Written in plain language, easy to understand, and available in 22 languages, this information explains the physical and mental effects of gs on a person. It tells e stories of people who have used gs and became addicted to them. If someone is trying to push a g on you. or if you are considering using a g or are already using it, or if you know someone using gs, check out this site so you have all the information you need to make an informed decision. The first segment, entitled The th About gs , gives a factual and concise overview of exactly what gs are, how they work and what they do. It gives the street names as well as the long - and short-term side effects of the mostmon substances. As a booklet, it is the rmended general handout for distribution in anymunity and to share with friends, family and others. Each individual g has its own section with a lot more facts and stories of users.

You can select which g you want to read about . The booklets and this site are resources for anyonekids, parents, teachers, counselors, law enforcement officers, youth workers, g prevention and rehabilitation specialists and health professionals. To read the booklets, just click on one of the covers above. The thirteen g information booklets are also free and can be ordered as a set, or each one of them can be ordered separately in any quantity. Click here to request The th About gs booklets. Sign in to receive rmendations (Learn more ) Femara (chemical name: letrozole) is an aromatase inhibitor approved by the U. S. Food and g Administration (FDA) to treat: postmenopausal women diagnosed with hormone-receptor-positive, early-stage breast cancer right after surgery (or possibly chemotherapy and radiation) to reduce the risk of the cancering back postmenopausal women diagnosed with hormone-receptor-positive early-stage breast cancer who have taken 5 years of tamoxifen to reduce the risk of the cancering back postmenopausal women diagnosed with advanced-stage or metastatic hormone-receptor-positivexA0breast cancerxA0 Femara wont work on hormone-receptor-negative breast cancer. Femara is a pill taken once a day. Most doctors rmend taking Femara at the same time each day. In 2011, the FDA gave approval for 11 pharmaceuticalpanies to make a generic version of Femara, which may go by the chemical name letrozole. You should not take Femara if you are breastfeeding, pregnant, trying to get pregnant, or if there is any chance that you could be pregnant. Femara may cause damage to developing embryos.

You should use an effective non-hormonal type of birth control -- such as condoms, a diaphragm along with spermicide, or a non-hormonal I. U.D. x2013 while you are taking Femara. Ask your doctor which type of non-hormonal birth control would be best for you, as well as how long you should use this type of birth control after you stop taking Femara. Benefits of Femara Two large studies have shown the benefits of Femara. The international BIG 1-98 trial, started in 1998,pared Femara to tamoxifen after surgery in postmenopausal women diagnosed with hormone-receptor-positive, early-stage breast cancer. The results showed that Femara was better than tamoxifen for: increasing the time before the canceres back in those who experience recurrence reducing the risk of the cancer spreading to other parts of the body The MA-17 study, conducted in Canada, looked at whether taking Femara for 5 years AFTER taking tamoxifen for 5 years (for a total of 10 years of hormonal therapy) could lower the risk of the cancering back in postmenopausal women diagnosed with hormone-receptor-positive, early-stage breast cancer. The results showed that Femara: reduced the risk of the cancering back reduced the risk of the cancer spreading to another part of the body compared to not taking Femara after 5 years of tamoxifen.

Side effects of Femara BecausexA0Femara lowers the amount of estrogen in the body, less estrogen reaches bone cells, which can lead to bone thinning and weakening and a higher-than-average risk of broken bones. This side effect can be very troubling for some women. If you have osteoporosis. your doctor may rmend that you take tamoxifen rather than Femara because of this possible side effect. Othermon side effects of Femara are: Some women may have other side effects while taking Femara: Some side effects may mean that youre having an allergic reaction to Femara. If you havexA0shortness of breath or chest pain. call your doctor immediately. xA0 How long do I take Femara In most cases, youll take Femara for 5 years. Doctors may rmend that some women take it for a longer period of time.

Does insurance cover Femara While costs vary, Femara can cost several hundred dollars per month. Femara also is available as a generic medicine, which is usually less expensive. If you have health insurance, check with your insurancepany to see if and how much of the cost of Femara is covered. If you dont have health insurance or your insurance doesnt cover the cost of Femara, ask your doctor or nurse about programs in your area that may be able to help. Also, ask your doctor about prescribing a generic version of Femara, which may be less expensive.

Novartis, thepany that makes Femara, has created the Femara My Next Steps program, which may be able to help with costs. In the United States, visit the Femara website for more information. You can also read the Breastcancer Paying for Your Care section for information on additional types of financial assistance and cost-lowering tips. Last modified on June 8, 2015 at 12:32 PMThe number of couples seeking consultation for infertility problems has steadily increased over the past decade, affecting 10x0221215 of the sexually active population. Abnormal semen production, a male factor infertility (MFI), is thought to be the cause of up to 50 of all infertilities in developed countries. There are potentially many different causes of male infertility, including hormonal, anatomical, and secondary to exposure to exogenous substances. In many cases of MFI, a definitive cause for abnormalities is never identified. Recently, the researchmunity has given greater attention to identifying causes of MFI ranging from gic Y chromosome microdeletions to mechanisms of environmental damage on sperm production.

Still evolving, is a clear understanding of how many pharmaceutical medications may cause MFI, which is often treatable and reversible. In this review we will outline the data regarding various pharmaceutical medications that have been investigated as possible causes of MFI. Keywords: Male Infertility, Medications, Pregnancy, Review, Sperm Introduction Causes of male infertility The number of couples seeking consultation for infertility problems has steadily increased over the past decade, affecting 10x0221215 of the sexually active population (1. 2 ). MFI contributes to approximately 50 of all infertility (2 x020134 ). MFI may is generally diagnosed through an abnormal semen analysis (SA). While the SA has multiple measured parameters, the most important are abnormalities in sperm count, ranging from fewer than normal sperm (oligospermia) to undetectable sperm (azoospermia), sperm motility, a condition known as asthenospermia, and sperm morphology (5. 6 ). The relative importance of each of these parameters, among other measures of semen quality, are constantly a subject of debate within the fertilitymunity (5. 7 ). A furtherplicating matter is the poor reproducibility of SA as an assessment tool for sperm quality and quantity. Indeed, data has shown that interjaculate coefficients of variation for sperm concentration and motility are estimated to be as high as 44 and 15 respectively (8 ). The causes of oligospermia or azoospermia can be divided into three distinct stages: Pre-Testicular, Testicular, and Post-Testicular, depending on the stage of spermatogenesis which is altered or impaired (9 ). Pre-testicular stage azoospermia results from the pituitary gland, part of the hypothalamic-pituitary-gonadal (HPG) axis, not producing proper hormones to stimulate the testes to produce sperm and is often due to an underlying endocrinologic abnormality (9 ). Testicular stage azoospermia is a failure of testicular function and Post-Testicular azoospermia is due to physical causes such as obsction (9 ). In the majority of MFI cases, a definitive cause for abnormalities is never identified (4 ). Pharmaceutical medications as well as recreational gs have been documented to impact semen producticular stages (2. 10. 11 ). In addition, some medications impair ejaculation and erectile function, as well as the ability to decrease libido (12 ). This review article will focus on the effects of certain pharmaceutical medications that may be associated with male infertility.

Anti-depressants Depression is amon disorder with a lifetime prevalence of approximately 16 (13 ). Rising levels of depressive disorders have in turn led to an increase in the use of prescription antidepressants, with more than 253 million prescriptions on record for 2010, the second most among all medications in the United States (14 ). The underlying cause of depression is still being investigated but is thought to include an over-activity of the emotion processing regions of ventral limbic and underactivity of the dorsal prefrontal cortex (15 ). Antidepressent medications, specifically selective serotonin reuptake inhibitors (SSRIs), are currently the treatment of choice for individuals suffering from depression (16 ). SSRIs have been implicated as a source of MFI. However, as the use of SSRIs has increased in recent years, few studies have evaluated the effect of SSRIs on MFI. Furtherplicating the picture is the fact that some studies have linked depression alone, without the use of medications, to altered testosterone levels, which could conceivably contribute to MFI (17. 18 ). However, several other studies have failed to demonstrate such a correlation (17. 19. 20 ). In 2006, researchers at Weill Cornell Medical College in New York published a case report that proposed a possible link between SSRIs and MFI (21 ). In 2010, the same group performed a retrospective study on thirty-five healthy male volunteers (22 ). The men were asked to provide semen samples at baseline and were then administered the g paroxetine, an SSRI, for a period of five weeks. Results indicated that the key sperm parameters of volume, concentration, motility or morphology were not adversely affected during the trial period. However, sperm integrity DNA analysis showed that mean sperm DNA fragmentation was significantly greater (30.3) in men after continued use of paroxetine whenpared to the baseline (13.8). Furthermore, self-reports from the subjects showed that 35 cited considerable changes in erectile function while 47 noted difficulties with ejaculation while taking paroxetine. Limitations of the study included a small sample size of healthy volunteers without baseline depression, a lack of placebo pill use in the control group, and the fact that fertility was not evaluated. Another retrospective trial has demonstrated an association between SSRI use and decreased sperm motility (23 ). Evidence also shows that SSRIs have a spermicidal effect in vitro (24 ). One group has actually described SSRIs as an acutely helpful therapy to improve increased ejaculate volume in men taking Sildenafil (25 ). While there have been several small trials that link SSRIs to semen abnormalities, the authors could not identify a large trial that definitively link SSRIs to MFI as defined by in ability to achieve pregnancy. As such, the data linking SSRIs to MFI are, at the present time, suggestive but not conclusive. A summary of the current evidence is offered in Table 1.

Sukraja 8211 Does Sukraja Work By Cheryl Powers on November 9, 2010 Last Modified: July 8, 2015 Sukraja Overview Sukraja . a product manufactured by India Herbs, is a male potency formula that uses the ancient Ayurveda method of treatment. Itbines a large number of traditional eastern herbs and botanicals that have long been used to treat male impotence . low sperm count and loss of libido. Sukraja promises to help all these conditions while supporting stamina, sperm count and strength ofasm. The Sukraja official website is very informative but contains so much information some consumers may find it overwhelming. While there are no customer testimonials, the product is doctor endorsed and the doctors responsible for developing the formula are profiled on the site. No clinical study results are shown for the Sukraja product, but certain ingredients have proven successful in independent tests.

The website includes an informative FAQ section and the product can also be ordered securely from the manufacturer, with free shipping offered on larger orders. A generous 4 month money back guarantee is provided and while results should be seen within one month, the optimal time of use is between 4-6 months. When purchased from the site Sukraja costs 60.00 for a one month supply, making it slightly more expensive than similar brands. Sukraja Product Details All ingredients for Sukraja are shown on the website and explained in full detail. The proprietary formula consists of 54 photonutrients, which are plantpounds that work synergistically in the body. The key ingredients are Tribulus, Masha, Piper Longum, Mucuna riens, Withania Somnifera and Asparagus Racemosus. Together, the formula helps support testosterone and growth hormone levels while detoxifying, calming the nerves and rejuvenating the prostate. Sukraja is taken on a daily basis at a dose of 2 capsules per day.

Supplement Alternative to Viagra The US Food and g Administration (FDA) and the pharmaceuticalpany Pfizer advised doctors last week about new warnings and information added to the labeling of the impotence g sildenafil (Viagra). The changes have been made in response to reports of cardiovascular adverse events in patients using the g and nitrate heart gs. According to the FDAs last surveillance update, there were 123 deaths worldwide reported from March through July 1998 for patients taking sildenafil. Of the 69 men who died in the US, 74 had one or more risk factor for cardiovascular or cerebrovascular disease, the agency said. Dr. Mercolasment: On March 27, 1998 the FDA approved Viagra. I have warned readers of this newsletter on three previous occasions of the dangers of this g and I would like to offer some practical alternatives.

Much of this information is taken from the excellent newsletter that Dr. Ward Dean (Vitamin Research News Vol 12. 9, November 1998) publishes. Viagra was initially investigated as a potential anti-angina medication, based on its ability to release nitric oxide and increase blood flow to the heart. Viagra failed as a heart medication, but London researchers became excited when many of the men in the clinical trials reported the frequent occurrence of unaccustomed erections and improved sexual performance. Five years after this serendipitous finding, Viagra was granted approval as a treatment for men suffering from difficulty in achieving erection. In the two months following its release, over one million prescriptions were issued, making it one of the most successful gs ever introduced. Viagra may even provide similar benefits, enhancing sexual sensation andasmic enjoyment, for women and the g is now in clinical trials for that purpose.

Viagra Alternatives. Vascular smooth muscle cells surround arteries and arterioles, contracting and relaxing the arteries to regulate blood pressure. The given state of smooth muscle cells, and their effect on blood pressure, understandably have a profound effect on the male sexualan. Normally, in the presence of sexual stimulation, blood flow is directed into pockets known as the corpus cavernosum, contained within the shaft of the penis. The resulting inflow of blood leads to the enlargement and stiffening of the penis. This enement is triggered by a short-lived neurotransmitter. Nitric oxide, synthesized from the oxidation of the amino acid arginine, activates an enzyme that manufactures cyclic guanosine monophosphate (cGMP) which is a biochemical signaling enzyme. Under normal circumstances, cGMP directs the smooth muscle cells to relax, leading to the dilation of the penile arteries. However, immediately following release of nitric oxide and production of cGMP another enzyme, another enzyme, cGMP phosphodiesterase type 5 (PDE-5), is activated.

PDE-5s main activity is to destroy cGMP almost as fast as it is formed. The result of this breakdown of cGMP by PDE-5 is a rapid decrease in smooth muscle relaxation and a loss of blood flow to the penis. Subsequently, the penis returns to a flaccid state. Unfortunately, as we age, cellular concentrations of cGMP decrease. Viagra works to achieve and maintain erections by enhancing the effect of nitric oxide and maintaining higher levels of cGMP. The way Viagra does this is to selectively inhibit the cGMP destroying actions of PDE-5. By blocking the actions of this enzyme system, cGMP is no longer broken down. This leads to elevated cGMP levels in the corpus cavernosum. This, in turn, prevents relaxation of the smooth muscle in the corpus cavernosum, increases blood flow to the genitals, and leads to stronger erections and intensified sensations.

Aside from the risk of dying and increasing the risk of retinal dysfunction, the biggest drawback to Viagra is the high cost, which can range upwards of 10 per pill. Fortunately, there is a safer, less expensive and more natural way to achieve many of the actions to Viagra. The key is L-arginine, the direct precursor to nitric oxide. In the 1990s, scientists discovered that L-arginine, a non-essential amino acid, monly found in the diet, is an oxidative precursor of nitric oxide. Under conditions in which nitric oxide is produced for a specified physiologic purpose, the concentration of L-arginine from which nitric oxide is formed, can be a limiting factor. Researchers at New York University School of Medicine gave L-arginine to a group of 15 men and found that six received benefit. Doses are around 1000 to 3000 mg per day. Sexual arousal occurs not just in the genitals, but in the whole body and especially in the brain. For men, it actually begins when the brain sends impulses down the spinal cord and out to the nerves that serve the penis. These impulses trigger the production of nitric oxide.

The neurotransmitter that causes the sexual message is acetlycholine (ACH). ACH also seems to control sexual behavior through its activity in the brain. For women, ACH is also a very important part of sexual function. With too little ACH, sexual activity goes down. ACH is involved in the buildup towardasm and the urethral and vaginal contractions that occur duringasm. One way to safely and effectively enhance ACH levels in the body is to take supplements of choline ( 1000 to 3000 mg) and vitamin B5 (500 to 1500 mg) so that the body will produce more ACH. Vitamin B5, also known as pantothenic acid, actually seems to enhance endurance by two routes. The first is by increasing ACH and the second is its role in the energy producing Krebs cycle. The choline and vitamin B5 are ideally taken about 20 to 30 minutes before sex in order to get the full effect.

Yohimbe and yohimbine is from a native tree in West Africa. For centuries a tea distilled from the inner bark of this tree has been used to amplify male virility and sexual prowess. Yohimbines primary path of action duplicates a key biochemical role in male erection. Yohimibine acts upon a specificwork of nerve cells called the alpha-2-adrengeric system. Yohimbine shuts down this system which increases the flow of blood through arteries into the penis while at the same time decreasing the blood flow from the penis through veins.

It also results in higher levels of ACH. Several controlled trials have been done with yohimbine and the response rate is about 40 percent. Spread the Word toFriends And Family By Sharing this Article.